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Detailed article Table of Contents from iPS cell immune cells cancer to patient care in the world's first iPS transplant implementation eyes of incurable patients, iPS cells in medical institutions of Kobe, more Cambridge University in state close to the fertilized egg is success iPS Institute: initial stimulus eliciting properties to pluripotency el cancer of essential technical TIPS body cells for reduction in, STAP cell conversion cultures from miscellaneous thoughts body cells for miscellaneous thoughts 2 STAP cells for state and found STAP cells similar to the occurrence early embryo fate conversion RIKEN, easily cancer rate than produced iPS cells universal cells in short-term low iPS siblings team, established Israel Institute of the screening method discovered novel human ground state naive pluripotent stem cells, somatic cell nearly 100% iPS to - identify the failure molecular el cancer carcinogenesis risk low iPS cell production Beijing, "confidence in the safety" iPS clinical without gene RIKEN project leader
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Induced pluripotent Huntington's disease monkey stem cells (rHD-iPSCs) were established by the overexpression of rhesus macaque transcription factors (Oct4, Sox2, and Klf4) in transgenic Huntington's monkey skin fibroblasts. The rHD-iPSCs were pluripotent and capable el cancer of differentiating into neuronal cell types in vitro and developed teratoma in immune compromised mice. We also demonstrated the upregulation of endogenous Oct4 and Sox2 after successful reprogramming to pluripotency in rHD-iPSCs, which was not expressed in skin fibroblasts. rHD-iPSCs also developed cellular features comparable el cancer to Huntington's disease (HD), including the accumulation of mutant huntingtin (htt) aggregate and the formation of intranuclear el cancer inclusions (NIs) paralleling neural differentiation in vitro. Induced pluripotent stem cells from transgenic HD monkeys open a new era of nonhuman primate el cancer modeling of human diseases. rHD -iPSCs that develop el cancer key HD cellular features and parallel neural differentiation can be a powerful platform for investigating the developmental impact on HD pathogenesis and developing new therapies, which can be evaluated in HD monkeys from whom the rHD-iPSCs were derived.
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