Mainly affects the joints throughout the body, and symmetry were multiple chronic proliferative ovu

Mainly affects the joints throughout the body, and symmetry were multiple chronic proliferative ovulation calendar synovitis, resulting joint capsule ovulation calendar and cartilage destruction, leading to joint stiffness deformity. ovulation calendar



Etiology and Pathogenesis
At present, many believe that this disease is an autoimmune disease, the initiating factor is unclear, it may be infected factor (such as viruses, mycoplasma or bacteria, etc.) into the human body, which contains some of the ingredients ovulation calendar (such as oligosaccharides or glycopeptide fragments ) uptake by synovial cells and incorporated into the intra-articular proteoglycan synthesis in synovial cells, but its structure changes with antigenicity. This not only allows the body to produce self-antigen antibody (IgG), but also lead to the Fc fragment of the IgG molecule structure change, the formation of new epitopes, so as to stimulate the formation of another antibody, i.e., rheumatoid factor (RF). The main component of RF in serum is IgM, also IgG, IgA and IgE and the like. IgM type RF met in 85% to 95% of the patients with rheumatoid arthritis, is an important indicator for clinical diagnosis. Immune complexes ovulation calendar of various immunoglobulin classes IgG RF and formed in the presence of blood circulation. RF and immunoglobulins can be synthesized within the joint and combined into immune complexes, the loop in RF-IgG complexes may also be deposited in the local tissue, which joints and extra-articular lesions in organs and tissues is closely related. Synovial the RF-IgG complexes may be fixed and start complement, C3a and produce C5a, attracts neutrophils and monocytes exudation. Neutrophils, monocytes and synovial cells (A-type cells) phagocytosis of immune ovulation calendar complexes described above, after being started and the synthesis ovulation calendar and release of lysosomal enzymes, including neutral proteases, collagenase, etc., and a variety of media, such as prostate , leukotrienes, IL-1, leading to destruction of synovial membrane and articular cartilage. IL-1 is the main medium of rheumatoid arthritis by the start of macrophages and synovial cells. IL-1 can synovial cells and chondrocytes synthesize and release collagenase and other proteolytic enzymes, and inhibits proteoglycan synthesis chondrocytes, which in turn is a start factor osteoclasts. Not only has RF, various immunoglobulin and complement etc. synovial ovulation calendar and tissue culture by immunofluorescence and also indicating that they may produce synovial B cells and plasma cells. Even in the case of the initiating factors (such as infection factor) has ceased to exist, RF continues to produce, resulting in recurrent inflammatory lesions become chronic inflammation.
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